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(1965) The catecholamine hypothesis of affectivedisorders: a review of supporting evidence. In such acase, where a reactive species produced during the metabo-lism of a chemical is responsible for its carcinogenicity, anappropriate cancer dose metric would be the lifetime averagedaily amount of metabolism in the target tissue divided bythe volume of the tissue, as was used in the PK riskassessment for methylene chloride.